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The research identified key molecules that help hair matrix and dermal papilla cells communicate and influence hair growth in cashmere goats.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

LRIG1 is linked to better survival in Merkel cell carcinoma.

MCPIP1 in myeloid cells is important for skin cancer development and healthy hair growth.

Alpha-MSH affects mitochondrial function, and MC1R mutations may increase skin aging.

The PML protein helps prevent skin cancer in mice.

AMPK activation may reduce melanoma risk in red-haired individuals.

MC-1R is present in skin cells and may help reduce inflammation.

pCLCA2 protein may help maintain skin structure and function.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

MC-1R in skin cells may influence inflammation and collagen production.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

MARCKSL1 is important for wound healing and could be a target to reduce scarring.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Merkel cell carcinoma is most likely to recur within two years of diagnosis, and factors like immune suppression, being over 75, and male sex increase this risk.

MC1R gene variations affect skin, hair color, UV sensitivity, and melanoma risk.

NDP-MSH protects brain cells and reduces inflammation in Parkinson's disease by activating MC1R and involving Tregs.

Oncogenic melanocyte stem cells can develop into melanoma similar to human cases.

New therapies and trials are needed for Merkel cell carcinoma, a tough skin cancer.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.

BMI1 is essential for preventing hair greying and maintaining hair color.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

BMAL1 controls skin cell growth and UV damage risk, peaking at night.

Researchers found a new gene variant linked to a rare bone disease, which doesn't always cause symptoms in carriers.

MRL/MpJ mice heal burns slower with more scarring and less tissue regeneration than BALB/c mice.

The mouse model is effective for studying ACL graft healing.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.