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The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Injecting lentiviral vectors into early gestation mice effectively targets skin stem cells for potential gene therapy.

LGD1069 effectively prevents breast tumors in mice without toxicity.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Rat hair follicle stem cells can be used to improve blood vessel growth in engineered skin.

A vitamin D receptor mutation causes rickets and affects immune responses.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Vitamin D receptor is essential for hair growth signaling.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Modified rat hair follicle stem cells can help create artificial hair follicles, blood vessels, and skin.

Vitamin D receptor helps prevent skin cancer and supports skin health.

The AVET system effectively delivers genes to human keratinocytes and may help treat skin diseases.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

cgVEGF164 boosts hair follicle growth in mice.

Vitamin D receptor may help protect against UV-induced skin cancer.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

A new mouse model helps study melanocyte cells using GFP expression.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.