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A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

The immune processes causing VKH and vitiligo are similar in dogs and humans.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

A new genetic mutation in the ODC1 gene causes developmental delay and other symptoms in a young girl.

Hair follicle samples effectively show how well the drug MK-0752 targets and engages with the Notch pathway.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

New mutations in KRT83 and KRT86 are linked to the hair disorder monilethrix.

A gene mutation causes monilethrix in a Chinese family.

Certain gene variations are found in people with polycystic ovary syndrome.

CMV infection increases the risk of GvHD after bone marrow transplants.

Different keratins have unique expression patterns in mouse skin cells.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

A new mutation in the HR gene is linked to a rare form of hair loss with limb deformities.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

MITF+ melanoma patients are more likely to have multiple melanomas and unique skin patterns.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Two new gene mutations cause a rare hair disorder.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Type I keratin genes are closely linked to the rex locus on mouse chromosome 11, affecting hair development.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

A variant in the KRT31 gene causes a rare hereditary hair disorder called monilethrix.