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The methods accurately measured brain androgens, showing most 5α-androstane-3α,17β-diol comes from outside the brain, while androsterone is both transported and made in the brain.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

The research found new potential mechanisms in mouse hair growth by studying RNA interactions.

The EDA pathway plays a key role in bone development by interacting with other signaling pathways.

NDRG1 protein helps infantile hemangioma, a common infant tumor, to grow, and its mismanagement by FOXO1 protein plays a big role in causing the tumor.

Synthetic 19-norprogestins promote bone cell growth and function through their non-phenolic metabolites.

The microenvironment, especially mechanical forces, plays a crucial role in hair growth and could lead to new treatments for hair loss.

Neuropilin 2 may be a biomarker for melanoma and affects melanocyte behavior.

Hair growth is controlled by cycles influenced by hormones and various signals.

Allopregnanolone likely doesn't influence ethanol's rewarding effects in these mice.

Neuroimaging suggests that treatments targeting brain steroids could help control epilepsy, especially types linked to the menstrual cycle.

Mechanical tension worsens keloid scars by activating inflammation and fibrosis pathways.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

There are two ways to start hair growth: one needs Stat3 and the other does not, but both need PI3K activation.

Targeting NMMHC IIA may help treat blood-brain barrier damage.

Blocking CYP17A1 enzyme may help improve certain brain function issues related to dopamine.

WNT signaling is crucial for starting mammary gland development.

Wnt-signaling is regulated differently in skin cells and immune responses during wound healing.

Stem cell therapy affects 15 key genes in rats with diabetes-related erectile dysfunction.

Chemotherapy can cause brain inflammation and damage, and understanding this process could help manage side effects.

Blocking the CXCL12–CXCR4 axis may help treat hair loss in alopecia.

Higher CRHR1 levels in AA patients lead to increased inflammation.

Human prostate cells produce more WISP1/CCN4 when there's not enough oxygen.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

PrPC is important for neural differentiation in cattle and mouse embryonic stem cells.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

Targeting Lgr5+ stem cells and Wnt signaling may effectively treat hair loss.

Mutant cells in hair follicles are influenced by their location and interactions with surrounding cells.

Understanding hair follicle communication can help treat hair loss.