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Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

DPP4, a molecule in skin, helps heal large wounds and regrow hair follicles when its levels are reduced.

The type of tumor suppressor gene lost affects the behavior of skin cancer.

Matrical tumors share a common growth mechanism involving the Wnt pathway and consistent PHLDA1 expression.

Skin aging and cancer development are influenced by the competition between stem cells.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

A special bioink with nanoparticles helps regrow hair by reducing inflammation and promoting hair growth signals.

Certain herbal compounds, especially from bitter melon, can inhibit cancer growth and promote hair growth by blocking PAK1.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Injecting lentiviral vectors into early gestation mice effectively targets skin stem cells for potential gene therapy.

Activating PPAR-γ signalling can protect hair follicle stem cells from damage caused by chemotherapy.

HPV type 56 can hide in hair follicles even without visible warts.

Targeting Keratin 17 may help overcome cancer therapy resistance.

A peptide from hair follicle stem cells can boost hair growth.

Blocking certain proteins in mouse skin can reduce and shrink skin tumors.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

Id2 gene helps keep hair follicle stem cells inactive.

Keratin 15 helps maintain skin cell growth and repair.

Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

The HIF-2α/ARNT complex is important for hair follicle development by controlling cell growth.

KRT72 gene helps form hair.

P63 is a marker for epidermal stem cells in rats.

Complex basal cell carcinomas need personalized treatment due to unique genetic mutations.

HPV infection is linked to better survival in advanced anal cancer, higher radiation doses improve survival, especially in HPV-negative patients, and prostaglandin E₂ pretreatment can protect mouse hair follicles from radiation damage.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

Suppressing ODC activity reduces tumor growth in hair follicles.

miR-370-3p slows sheep hair cell growth by blocking SMAD4.