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Early involution in Hirosaki hairless rats' mammary glands is linked to a unique modification of STAT5A.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

γδ T cells help with hair growth during wound healing in mice.

Blocking a specific protein signal can make hair grow on mouse nipples.

Mutations in the SHH pathway in certain skin cells can cause skin tumors and abnormal hair growth.

TACE/ADAM17 is essential for maintaining healthy hair and hair follicle stem cells.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Gene regulation evolved differently in mouse and chicken skin, but remained stable in their trunks.

TMPRSS2 helps viruses enter cells and protects the prostate from inflammation-related cancer.

Minoxidil does not work to inhibit lysyl hydroxylases in newborn mouse lungs.

Reducing neutrophils or inhibiting NETs improves wound healing in sickle cell disease.

Activating PKM2 and Wnt/β-catenin signaling together can potentially enhance hair growth and could be a treatment for hair loss.

Dutasteride showed some prevention of prostate issues but also had limitations, especially with high-grade tumors.

Genetic mutations that disrupt homocysteine breakdown lead to increased damage in mouse hair keratin.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

No consistent link between genotype and phenotype in 5-α-Reductase type 2 deficiency.

The spiny mouse can regenerate its skin without scarring, which could help us learn how to heal human skin better.

RNase L hinders hair follicle regeneration by altering immune signals.

Deleting the CDSN gene causes severe skin and hair problems, leading to death.

Retinoic acid and DMSO improve gene delivery to mouse skin for potential hair and skin disease treatment.

SMAD4 is crucial for muscle repair in young adults but not in aged mice.

A high-fat diet may weaken tongue structure by reducing certain protein genes.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Fat stem cells from diabetic mice can still help heal wounds.

DKK2 and SOSTDC1 together are necessary for the normal timing of the first regression phase in the hair growth cycle.

The Msx2 gene affects feather development in Hungarian white geese and a specific gene variation could indicate feather quality.

CD34 is a marker for isolating stem-like cells in mouse hair follicles.

Overexpression of ΔNLef1 in mouse skin leads to hair loss, cysts, and skin tumors.

Rodent spiny hair traits are due to genetic factors other than the Edar gene.