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The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Engineered bacteria can deliver antioxidants to protect skin.

Ceramide Synthase 4 is crucial for healthy skin barrier function.

Lichen planopilaris involves disrupted fat metabolism, increased scarring, and mast cell activity.

Substance P may play a role in the inflammation seen in keratosis follicularis spinulosa decalvans.

Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

Canine hair follicle cells show stem cell properties, aiding hair growth.

Understanding skin stem cells and their regulation is key to improving skin healing and treating disorders.

Portable point-of-care testing can improve quick and accurate genetic disorder detection.

The shape of fibrous scaffolds can improve how stem cells help heal skin.

Intralesional cidofovir may be a viable alternative treatment for SCC.

Cysteines in wool fibers are accessible and form important disulfide bonds.

SKO-derived SKP-like cells may help with hair regeneration and skin restoration.

3D cell spheroids can help reduce scars by delivering therapeutic vesicles.

CCCA involves immune response and metabolism issues, suggesting new treatment options.

A new ethical skin model using stem cells offers a reliable alternative for dermatological research.

The hedgehog signalling pathway is key in skin development and basal cell carcinoma, offering insights for prevention and treatment.

Three genes, BMP4, POSTN, and WNT5A, may help treat keloids.

SGK3 is essential for proper hair growth and health.

Insurers opposed ASCO's recommendation for a single cancer care pathway program.

ATP-dependent chromatin-remodeling complexes are crucial for gene regulation, cell differentiation, and organ development in mammals.

Patients with Systemic Sclerosis have much higher levels of GDF-15, which could help predict organ involvement and guide treatment.

The technique effectively shows how human skin and hair cells form into ball-like structures.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.

A protocol was developed to create 3D skin models from adult diseased cells to study Small Fiber Neuropathy.

Mouse skin cells can become sperm-like cells in the lab.

CCCA may be a fibroproliferative disorder, and anti-fibrotic therapies could help.

A mutation in the Sgkl gene causes defective hair growth in mice.