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Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

GLI2 increases follistatin production in human skin cells.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

A parasite molecule can speed up skin healing and reduce scarring.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Deleting Gpr54 speeds up hair growth and regeneration.

K10 may not prevent tumors as previously thought and might increase benign tumor risk.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

The enzyme is crucial for skin cell development and can be activated without proteolytic activation.

EGF affects hair and skin development.

Gene therapy has potential as a future treatment for Hutchinson-Gilford progeria syndrome.

A man developed skin lesions as a side effect of a gamma secretase inhibitor used for treating a tumor.

Overexpression of R-spondin 3 leads to sparse hair and impaired hair regeneration.

Overexpressing IGF-1 in mice leads to skin abnormalities and tumors.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Lack of functional CYLD in mice leads to early aging and cancer.

Certain genetic changes in the KRT82 gene may cause patchy skin in New Zealand rabbits.

miR-325-3p can slow down brain tumor growth by targeting FOXM1.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

SK2 channels help control sensory signals in rat muscle spindles and hair follicles.

Certain gene variations in EGF and EGFR may increase the risk of alopecia areata in Koreans.

A mutation in the hHb3 gene is linked to the hair disorder monilethrix.

EGFR helps control how hair grows and forms without needing p53 protein.

A new genetic mutation linked to Hutchinson-Gilford progeria syndrome was found in China.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.