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The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

High levels of human keratin 16 in mice cause skin lesions and abnormal skin development.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Sonic hedgehog signaling is crucial for normal wound healing.

Sox2 controls hair color by affecting pigment production in hair follicles.

Hyaluronan from Has2 is important for proper wound healing and hair follicle development.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Reducing SOX2 in colorectal cancer cells decreases tumor growth and self-renewal.

Different melanocyte types in hair follicles either survive or die during the catagen phase.

The HoxC gene cluster and its enhancers are essential for developing hair and nails in mammals.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

HSL treatment speeds up hair growth and reduces oxidative stress in hair follicles of ob/ob mice.

Antizyme slows skin tumor growth by reducing cell growth in mice.

ODC transgenic mice can model human hair loss with skin lesions.

Increased 14-3-3 proteins may block hair cycle regression, causing hair loss.

β-estradiol slows hair growth in mice.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

The spiny mouse regenerates ear tissue asymmetrically, with gene expression differences possibly explaining its unique healing abilities.

Mouse epidermal neural crest stem cells can become various cell types and are easily obtained from hair follicles.

Understanding normal hair follicle development helps analyze abnormalities in mutant mice.

Defects in certain proteins cause major heart abnormalities during early development.

The PML protein helps prevent skin cancer in mice.