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Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Natural α-hydroxyl acids cause skin exfoliation by activating TRPV3 channels.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

TECAR therapy helps with pain relief, reduces inflammation, relaxes muscles, heals tissues, and is safe and effective for many conditions.

A woman with a unique syndrome similar to TRPS has a genetic change near the TRPS1 gene, affecting its regulation.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Vav2 and Vav3 proteins help control skin stem cell numbers and activity in both healthy and cancerous cells.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Deucravacitinib led to full hair regrowth in a severe alopecia areata patient.

Activating TRPV3 reduces skin oil production and increases inflammation, potentially causing dry skin issues.

NIR-II imaging effectively tracked stem cells that helped repair facial nerve defects in rats.

Rat vibrissae structure relates to their sensory function.

Recognizing RSCC is crucial due to its aggressive nature and high risk of poor outcomes.

The new method combining dermoscopy and Reflectance Confocal Microscopy is more effective for evaluating vitiligo.

VR1 signaling can inhibit hair growth by affecting cell processes and increasing hair growth inhibitors.

TRPA1 and TRPV1 channels don't affect mechanotransduction in rat sinus hair follicles.

TRPV4 slows hair growth by affecting hair follicle cells.

The sensor accurately measures thallium ions in solutions with high selectivity.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

A new method allows for controlled, long-lasting delivery of retinoic acid through the skin with less inflammation.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

VDAC2 promotes cell death in cashmere goat hair follicles through the P53 pathway.

The new delivery system improved raloxifene's skin absorption and effectiveness against cancer cells.

TTD patients don't have a higher skin cancer risk because their main issue is with transcription, not DNA repair.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

A person with a specific gene mutation had extra teeth, unique jaw and hair features not seen before in this condition.