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Wnt4 protein makes the outer skin layer thicker in burn wounds by turning on a specific healing pathway and loosening the connections between skin cells.

Allergic contact dermatitis may promote hair growth by activating hair follicle stem cells.

Activating TLR3 boosts autophagy gene expression in skin cells.

The new antibody, TYHF-1, specifically targets certain hair-related structures.

PDCD4 is important for controlling skin cell growth and healing.

IL-27 may help prevent hair loss by creating immune-suppressing cells.

IL-13 protein is much higher in the skin of atopic dermatitis patients than in healthy skin.

Activating TLR3 may help produce retinoic acid, important for tissue regeneration.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

Endothelial TLR2 is crucial for timely wound healing, but HFSC TLR2 is not needed.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

β‐thymosins, especially thymosin β4, help in heart protection, wound healing, and hair growth by stabilizing actin in cells.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

Dandruff is linked to changes in the immune system of hair follicles and skin.

γδTregs can protect hair follicles from alopecia areata and may help regrow hair.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

Using low-dose IL-2 to increase regulatory T cells might be a safe way to treat type 1 diabetes without severe side effects.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

TNFα helps grow and maintain liver cells in 3D culture for a long time.

CDK4 levels affect the number of hair follicle stem cells in mice.

A new mouse model effectively mimics vitiligo for research and drug testing.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.

Myeloid cells can turn into skin and hair cells to help heal wounds.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.