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Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Androgens reduce THY1 in skin cells, leading to less fat, more fibrosis, and worse healing in males.

Injecting recipient splenocytes into donors' thymus can prevent graft-versus-host disease.

Heat shock proteins help basal cell carcinoma grow by responding to inflammation signals.

Skin cells and immune cells change in a skin condition called hidradenitis suppurativa, and a certain treatment can improve these changes.

Blocking the CXCL12–CXCR4 axis may help treat hair loss in alopecia.

Chronic refractory alopecia areata has more skin-resident memory T cells, and JAK inhibitors may help reduce them.

Type 2 cytokines IL-4 and IL-13 increase skin's vulnerability to Staphylococcus aureus, which could be important for treating atopic dermatitis.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

Treg cells help repair and regenerate tissues by interacting with local cells.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Certain immune cells, when activated by specific signals, can encourage hair growth.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

The CD44-CD49d complex boosts T cell activation and survival in autoimmune disease.

Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Reduced AhR signaling in HS tunnels leads to persistent inflammation and microbial imbalance.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Certain immune cells may cause hair loss by reacting to stressed hair follicles.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

Type 2 immunity helps control mite growth in hair follicles, preventing damage.

Interleukin levels are higher in alopecia areata patients but don't predict disease severity or duration.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

Only one K16 gene on chromosome 17 makes a functional keratin protein.

CD80 on skin stem cells helps expand Treg cells to aid wound healing.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.