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The research suggests that a specific skin gene can be controlled by signals within and between cells and is wrongly activated in certain skin diseases.

Autophagy is important for determining the protein makeup of hair.

Targeting PTEN can improve healing in venous leg ulcers.

Loss of the Y chromosome and UTY gene activity increases cancer risk in men.

Activating Wnt signaling improves the efficiency and safety of creating stem cells.

NFATc1 controls hair stem cell activity, affecting hair growth and could be a target for hair loss treatments.

Foxc1 helps keep hair follicle stem cells inactive, preventing hair loss.

The HR protein's role as a repressor is essential for controlling hair growth.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

The research found that a protein called caveolin-1 is reduced in psoriasis, but reintroducing it can help alleviate some psoriasis symptoms.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Deleting the CDSN gene causes severe skin and hair problems, leading to death.

Hoxc8 gene helps start mammary gland development by controlling specific signals.

UVB exposure affects retinoid metabolism, which is important for skin cancer progression and treatment.

Aging hair follicle stem cells can be rejuvenated by inhibiting Cdc42.

Certain nutrients like keratin, egg yolk, and fish collagen may help pets regrow hair.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

A gene variant causes a skin and hair disorder by disrupting protein balance.

Covering a wound can stop hair growth by promoting scarring, but boosting a process called Wnt signaling can help hair grow back even when the wound is covered.

Without keratin 10, there's more growth and development of oil-producing skin cells.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Autophagy changes the protein makeup of hair.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

A cat's poor wound healing was linked to a genetic deletion in the COL5A1 gene.

Disrupting Notch signaling in blood vessels increases scarring during wound healing in mice.

HoxC genes are crucial for normal hair and nail development.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.