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Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Mutant stem cells adapt their metabolism differently to outcompete normal cells in the skin.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

miR-199a-3p controls hair growth and is linked to alopecia areata.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

Wnt signaling is crucial for Hedgehog-driven skin tumor growth.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

The keratin tail is crucial for skin structure and function.

JAK/STAT1 pathway causes hair loss during chemotherapy by reducing Shh in hair follicles.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

Urotensin II increases growth and VEGF production in rat skin cells by turning on the Wnt-β-catenin pathway.

CuATSM speeds up wound healing and reduces scarring.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

Deleting the scd1 gene in mice prevents obesity by disrupting skin lipids and improving heat regulation.

Quercitrin may help treat Alzheimer's by boosting brain cell signaling without causing tumors.

Special gut cells help stem cells move to and fix injured areas by activating a specific signaling pathway.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Matriptase is crucial for keeping epithelial tissues healthy and functioning properly.

Both main and alternative Wnt signaling are important for regrowing rodent whisker follicles.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

CCCA involves immune response and metabolism issues, suggesting new treatment options.