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UVB exposure in human skin causes macrophages to produce more IL-10 and less IL-12, leading to immunosuppression.

Hair follicles help skin immune recovery after UVB exposure.

Combining glycyrrhizin and UVB light is most effective for improving vitiligo.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Disrupting Bcl-xL in mice reduces skin cancer risk.

Sebaceous lipids are crucial for keeping skin and eyes healthy in mice.

Eclipta prostrata extract protects skin cells from UVB damage and has strong antioxidant properties.

Low-sulfur hair syndrome can cause UVB sensitivity and testicular failure.

Egg shell membrane hydrolysates can reduce wrinkles and improve skin health.

Twist1 is crucial for UVB-induced skin cancer development.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

15d‐PGJ2 cream protects against UVB-induced skin damage in mice.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Prolonged UVB exposure causes significant skin changes and damage in rats.

Sinapic acid may protect skin cells from UV damage and reduce inflammation, making it a promising anti-aging cosmetic ingredient.

Dead Sea water and magnetized saline water can protect skin from UVB damage by activating autophagy.

UVB exposure increases skin proteins for retinoic acid synthesis and shifts their location, possibly affecting skin repair.

Cis-UCA can form new compounds without enzymes, and sulphide donors reduce its UVB-induced toxicity in skin cells.

Tanning ability is linked to specific DNA changes in skin genes.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

Combining UVB irradiation and anti-CD154 antibody improves hair follicle transplant survival.

UVB exposure affects retinoid metabolism, which is important for skin cancer progression and treatment.

Lysine carboxymethyl cysteinate (LCC) protects skin from UVB damage by activating autophagy.

Camellia sinensis seed flavonoids can reduce skin inflammation and damage from UV rays.

Gynostemma pentaphyllum and Damulin B help prevent skin aging from UVB by reducing oxidative stress and improving skin health.

Centella asiatica extract taken orally can reduce skin aging from UVB exposure.

Exosomes from stem cells can reduce UV-induced skin damage by lowering inflammation and oxidative stress.

Exosomes from hair stem cells can reduce skin aging from UVB exposure.

Deleting Twist1 in skin cells reduces UVB-induced skin cancer risk.