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Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

CMV infection increases the risk of GvHD after bone marrow transplants.

Monoclonal antibodies LT-1, LT-2, and LT-7 help diagnose certain blood cancers.

γδ T cells help with hair growth during wound healing in mice.

Regulatory γδ T cells help protect hair follicles from alopecia areata and promote hair regrowth.

The new ddPCR method reliably detects unwanted viruses in CAR-T cell products, ensuring their safety for patients.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

BST2 protein and certain T cells increase in early alopecia areata.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

Dermal IgA deposition without symptoms is rare in Dermatitis herpetiformis risk groups.

IFN-γ production by CD4 T cells is crucial for causing alopecia areata.

γδTregs can protect hair follicles from alopecia areata and may help regrow hair.

Dual TCR Treg cells are common in mouse tissues and vary by location.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

IL-17-producing γδ T cells help improve bone healing.

Donor lymphocyte infusions effectively treated leukemia relapse but caused vitiligo and alopecia areata.

CD4 T cells need IFN-γ to cause hair loss in alopecia areata.

The CD4 protein may play a role in the behavior of certain skin cells, affecting their growth, movement, and differentiation.

Graft-versus-host disease is a complication where donor immune cells attack the recipient's body, often affecting the skin, liver, and gastrointestinal tract.

DEC cells show promise as a safe and effective treatment for Duchenne muscular dystrophy.

Special cells can help regrow hair in alopecia areata.

IL-1 signaling is crucial for hair follicle stem cell growth and wound healing.

HTLV-1-associated lichenoid dermatitis (HALD) is linked to an immune response against HTLV-1-infected cells.

Pemphigus vulgaris involves specific immune cells and B cells that produce antibodies causing skin blisters.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.