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Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

VDR regulation varies by tissue and is crucial for its biological functions.

Nocturnal calcium infusions improved a boy's severe rickets without causing hair loss.

Hairless protein can block vitamin D activation in skin cells.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

The vitamin D receptor can work without its usual activating molecule.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Vitamin D receptor is crucial for normal hair growth and preventing hair loss.

Vitamin D and calcium are essential for effective wound healing and hair growth.

The HR protein's role as a repressor is essential for controlling hair growth.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

A girl had rickets due to a gene mutation affecting vitamin D response.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Key genes and pathways influence cashmere production in goats.

High CRP levels could indicate vitamin D deficiency in people with alopecia areata.

Vitamin D and its receptor help regulate skin functions like cell growth, immunity, and hair cycle.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

ΔNp63α helps control a protein that stops cancer cells from spreading.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.