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The immune processes causing VKH and vitiligo are similar in dogs and humans.

Scientists found a specific group of itch-sensing nerve cells in mice important for feeling itch but not for sensing heat or touch.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Mice with the 'lanceolate hair' mutation have abnormal hair and skin similar to human Netherton's syndrome.

Hirosaki hairless rats have sparse, twisted hair due to missing hair keratin genes.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

B6J mice live longer before 24 months, but B6N mice live longer after; both strains show weight gain, increased food and water intake, and health issues as they age.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

MED1 affects skin wound healing differently with age, speeding it up in young mice but slowing it in older mice.

CD44 variant changes start alopecia areata, but don't maintain it.

The spiny mouse is a unique menstruating rodent that can help us understand menstruation and reproductive disorders.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

The Agouti gene influences pigmentation and may have a developmental role in deer mice.

Testosterone causes hair loss in AGA mice, which are good for testing baldness treatments, and both minoxidil and cyproterone acetate can prevent this hair loss.

Hearing decline in SAMP8 mice starts before outer hair cell loss and may be linked to other changes.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Researchers can now observe live cell processes in the Drosophila midgut for extended periods.

Recent advances show zebrafish can model anemia, Alx4 affects craniofacial and hair development, and mTORC1 is crucial for retinal development.

Alopecia areata can be triggered by specific immune cells without genetic or environmental factors.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

MRL/MpJ mice heal burns slower with more scarring and less tissue regeneration than BALB/c mice.

The K5 promoter controls gene expression in skin cells, with specific DNA segments crucial for targeting and regulation.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

Alopecia areata is an autoimmune disease that targets hair follicles.

Androgen loss may speed up hair greying.