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The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D receptor FokI gene variation is not linked to alopecia areata.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Vitamin D receptor helps prevent skin tumors.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Vitamin D receptor and mediator 1 are crucial for healthy skin and hair growth.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Vitamin D helps regulate calcium levels in the body.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Vitamin D and its receptor are important for hair and scalp health and may help treat hair loss.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Vitamin D and its receptor help regulate skin functions like cell growth, immunity, and hair cycle.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Key genes and pathways influence cashmere production in goats.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Vitamin D receptor is crucial for hair health and may help treat hair loss.

Vitamin D may help reduce severity and relapse of alopecia areata.

The Tru9I variant in the VDR gene may influence alopecia areata risk and vitamin D levels.