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δ-GABAA receptors affect alcohol consumption based on the estrous cycle and influence movement regardless of the cycle.

The HPV type 11 region activates hair-specific gene expression in mice.

VR1 signaling can inhibit hair growth by affecting cell processes and increasing hair growth inhibitors.

Cannabidivarin (CBDV) may help brain cell growth and survival through the TRPV1 receptor.

The environment around the time of conception can change the VTRNA2-1 gene in a way that lasts for years and may affect disease risk.

Estrogen increases blood vessel growth factor production, while testosterone blocks this increase.

DHT enhances androgen receptor activity more than testosterone, and MAGE-11 influences this activity through specific interactions.

Activin B can boost hair growth by promoting cell proliferation and cell cycle progression.

Trps1 is essential for proper hair follicle development.

Injecting lentiviral vectors into early gestation mice effectively targets skin stem cells for potential gene therapy.

Androgen receptor activity is influenced by hormones, co-factors, modifications, and mutations.

Certain interactions help prepare the androgen receptor for pairing and activation, which is important for its role in development and disease.

Lentiviral vector effectiveness in skin is influenced by external factors, not receptor availability.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

Carvacrol activates the TRPV3 channel, which may help design new skin treatments.

Activating TRPV3 reduces skin oil production and increases inflammation, potentially causing dry skin issues.

Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

Activating TRPV4 in skin cells helps regrow hair in mice, possibly offering a treatment for hair loss.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Rat hair follicle stem cells can be used to improve blood vessel growth in engineered skin.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Immortalized rat dermal papilla cells can still induce hair growth.

Activating TRPV3 channels stops hair growth by killing hair follicle cells.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

VEGF promotes hair follicle cell growth through the VEGFR-2/ERK pathway.

A new method allows for controlled, long-lasting delivery of retinoic acid through the skin with less inflammation.