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Vitamin D3 speeds up skin healing by helping skin stem cells grow and develop.

The skin produces and uses vitamin D for bone health, cell growth, and immune function.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

Vitamin D receptor is crucial for bone health and mineral metabolism.

Vitamin D may help treat hair disorders.

Vitamin D receptor may help protect against UV-induced skin cancer.

Loss of keratin K2 causes skin problems and inflammation.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Kevis P.F. may help with hair care when used with other products.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Vitamin D receptor actions without binding are crucial for healthy skin and hair.

Vitamin D is made by the skin, helps control various body functions, and affects skin health and immunity.

Vitamin D and its receptor help regulate skin functions like cell growth, immunity, and hair cycle.

Vogt-Koyanagi-Harada disease affects vision and skin, mainly in people with darker skin, and is treated with steroids and immunosuppressants.

Vitamin D helps treat psoriasis by promoting skin cell differentiation and reducing growth.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

The vitamin D receptor can work without its usual activating molecule.

A vitamin D receptor mutation causes rickets and affects immune responses.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Vitamin D receptor is essential for healthy hair growth.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A patient with pemphigus vulgaris improved significantly after treatment for an additional viral skin infection.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Kenikir leaf extract nanosuspension may help inhibit breast cancer cell growth.

Vitamin D protects skin cells from damage caused by captopril.

K25, K27, and K28 are found in all inner root sheath layers of hair, while K26 is only in the cuticle.