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Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Wnt-signaling is regulated differently in skin cells and immune responses during wound healing.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

DHT reduces a cell's ability to promote hair growth, while 3D culture without DHT improves it.

Females have higher estrogen receptor levels in hair than males, and these levels decrease in white hair compared to black hair.

The study suggests that a specific gene variation and higher gene activity are linked to increased baldness in Egyptian men.

Red light and LED treatments help hair grow by activating a specific cell signaling pathway.

Wnt10b helps blood stem cells grow after injury.

Changing Wnt signaling can lead to more or less hair growth and might help treat hair loss and skin conditions.

miR-22 helps skin cancer grow and spread by activating specific cell signals.

Certain nutrients like keratin, egg yolk, and fish collagen may help pets regrow hair.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

A genetic change in the EDAR gene causes the unique hair traits found in East Asians.

RANK-RANKL signaling is essential for hair growth and skin health.

Non-canonical Wnt signaling helps intestinal stem cells move to injury sites for tissue repair.

Parathyroid hormone-related protein helps control hair growth phases in mice.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

The PTGER2 gene is highly active in Cashmere goat skin and its activity changes with the hair growth cycle.

Decreased IGF-1R expression may contribute to sacrococcygeal pilonidal sinus development.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.