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Specific immune cells and pathways contribute to hair follicle inflammation and hair loss, suggesting potential treatments for lichen planopilaris.

MSCs help rejuvenate skin by promoting cell growth and reducing inflammation.

Blocking certain immune signals can reduce skin damage from radiation therapy.

HTLV-1-associated lichenoid dermatitis (HALD) is linked to an immune response against HTLV-1-infected cells.

Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

Expanding regulatory T cells may help treat alopecia areata by reducing harmful immune cells.

EH-MSCs may help treat hair loss by reducing inflammation.

ILC1 cells contribute to hair loss in alopecia areata.

A new compound from Royoporus badius activates immune cells and induces inflammatory responses.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

DNMT1 helps turn hair follicle stem cells into fat cells by blocking a specific microRNA.

Langerhans' cells are not involved in hair depigmentation in these mice.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

Basal cell carcinomas need extra mutations to grow from small to large tumors.

Cells that move well may improve hair loss treatments by entering hair follicles.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

MAIT cells may help fight COVID-19 but also contribute to severe inflammation.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Certain NK cell changes in blood may indicate alopecia areata progression.

Sex and race affect immune responses and treatment outcomes in Hidradenitis suppurativa.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

People with alopecia areata have fewer IL-10 producing immune cells, which might contribute to the condition.

IL-15 promotes hair growth and protects hair follicles.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Androgens reduce macrophage receptor expression, affecting immune response, while antiandrogens counteract this effect.

Alopecia areata patients have more activated T cells in their blood, which may help in developing treatments.