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Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Jag2 is essential for proper skin cell differentiation and organization.

mEphA1 receptor tyrosine kinase is important for skin and hair development and may play a role in certain diseases.

Ceramide synthase 4 is essential for maintaining skin barrier health.

Different PADI isoforms help cells develop diverse functions.

Cepharanthine may help treat lung fibrosis by affecting certain immune cells.

TCDD reduces EGF receptors in the liver, affecting growth and development.

Blocking CXXC5 speeds up diabetic wound healing by improving blood vessel growth and skin repair.

Targeting NCoR1 can help treat heart enlargement and dysfunction.

Ivacaftor can protect against noise-induced hearing loss by reducing oxidative stress.

Basonuclin 2 is vital for the development of facial bones, hair follicles, and male germ cells in adult mice, and its absence can lead to dwarfism and abnormal follicles.

ARQ-234, a protein designed to treat atopic dermatitis, shows increased effectiveness in early testing.

Krox20 is important for maintaining stem cells in the skin and affects hair growth and color.

Calcium is crucial for skin development and healing.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

EDA and EDAR are important for hair follicle development in cashmere goats and affect other related genes.

A specific gene mutation causes a severe skin disorder in a family.

A cluster of 21 keratin-associated protein genes important for hair growth was found on human chromosome 21.

Corin helps control salt and sweat release in sweat glands.

Extracellular vesicles from mammary cells help heal skin wounds effectively.

CARB is a strong barrier in human hair that prevents dye penetration.

The Ysc84/SH3yl1 protein family is important for cell movement and the process of taking in materials by interacting with actin and cell membranes.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

Tissue-engineered skin substitutes can model junctional epidermolysis bullosa and may help develop gene therapy.

The new model helps understand and develop treatments for genetic skin disorders like AEC.

Certain genetic variants impair enzyme activity, contributing to non-classic congenital adrenal hyperplasia.