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Minoxidil 2% is more effective for female hair loss, but botanicals have fewer side effects.

Long-term data and a team approach are needed to manage chronic side effects from cancer treatments.

Bioprinting is improving skin models for better testing of skin diseases without using animals.

NMN supplements may improve hair quality and appearance in middle-aged women.

Allopregnanolone may help reduce inflammation and symptoms related to post-finasteride syndrome.

Aligned membranes improve wound healing by reducing scars and promoting skin regeneration.

Allopregnanolone can reduce gut inflammation and normalize neurotransmitter levels after finasteride withdrawal.

The cuneate nucleus has two main neuron types: relay neurons and interneurons.

Voriconazole can cause serious side effects, especially in long-term use.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

Researchers created a new mouse model for studying scleroderma.

Scientists made a mouse that can be made to lose hair and then grow it back.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Brindled mice show abnormal catecholamine neuron development due to copper deficiency.

The study found that the Marangoni effect causes the uneven wetting of surfactant-coated hair due to the surfactant moving into the water.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

New mouse models help study melanocytic cells for melanoma research.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Mouse models are essential for studying and improving genetic traits in agriculture.