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The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Monoclonal antibody B72.3 selectively reacts with certain dog tissues, mainly in the gastrointestinal and respiratory tracts.

Targeting NMMHC IIA may help treat blood-brain barrier damage.

High CRP levels could indicate vitamin D deficiency in people with alopecia areata.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Human and mouse TGase3 enzymes are similar but differ near the activation site, crucial for their function in skin and hair development.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Ku80 is a key receptor for Thymosin β4, affecting cell migration and wound healing.

Gene variation affects prostate issues and hair loss.

TIP39 and PTH2R help control calcium levels and skin cell development.

Blocking certain receptors in the lungs might help treat a specific type of asthma.

20(S)-Protopanaxadiol-aglycone may help prevent and treat prostate cancer by reducing androgen receptor activity.

A new genetic mutation in the CDH3 gene causes hair loss and eye problems in young people.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

GPCRs are vital for wound healing by affecting cell growth and immune response.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Certain defective glucocorticoid receptor mutants move faster inside cell nuclei and work less effectively.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

The enzyme system in hair follicles is similar to the liver's and is affected by certain inhibitors.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

α3β1 integrin is essential for skin stability and proper wound healing.

DMBT1 and galectin-3 may help suppress epithelial skin cancer.

Finasteride reduces certain behaviors caused by D1-like receptor agonists but not by D2-like receptor agonists in mice.

Vitamin D and its receptor help regulate skin functions like cell growth, immunity, and hair cycle.

Phospholipids help plant proteins move by regulating receptor interactions.