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Two new enzymes, PAD-R11 and PAD-R4, were cloned and showed activity, with PAD-R11 resembling epidermal enzymes.

Overexpression of COX-2 in mice skin causes abnormal skin and hair development.

Inhibiting ACE2 improves skin regeneration during tissue expansion.

Foxc1 helps keep hair follicle stem cells inactive, preventing hair loss.

HT-scCAT-seq helps understand gene regulation in embryonic skin development.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

Nicotinic acid reduces excessive oil production in skin cells by activating a specific receptor, which could help treat acne.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Ornithine decarboxylase is crucial for human hair growth and the hair cycle.

Vitamin D receptor helps control skin cell growth and hair development.

Suppressing ODC activity reduces tumor growth in hair follicles.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.

Ornithine decarboxylase is crucial for hair growth regulation in mice.

BMI1 is essential for preventing hair greying and maintaining hair color.

WWP2 is crucial for tooth development in mice.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.

Both SMS1 and SMS2 are crucial for normal hair growth and cycle in mice.

The hairless (hr) gene is essential for normal hair follicle function and its mutation leads to hair loss.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

hHb1, hHb3, and hHb6 mRNAs start expressing at the same time in hair follicles.

RNase L hinders hair follicle regeneration by altering immune signals.

Enhancing Tregs can protect against alopecia areata.

Msx-2 gene removal speeds up skin wound healing in mice.

Hair follicle stem cells prevent melanocyte stem cells from differentiating by controlling retinoic acid levels.

Increasing SSAT makes skin more prone to cancer.

Mutations in Far2 mice cause hair loss due to sebaceous gland issues.