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A patient with Ivemark syndrome developed mixed type vitiligo after a hepatitis C infection, showing different treatment responses and immune cell involvement in the skin.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Certain cytokines may cause or be affected by immune skin diseases, suggesting possible new treatments.

LncRNA RP11-818O24.3 may help treat hair loss by boosting hair follicle stem cell growth and differentiation.

Higher IL-17 and IL-23 levels may worsen alopecia areata.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Targeting specific cell interactions may help treat skin fibrosis.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

Visceral leishmaniasis can mimic rheumatoid arthritis symptoms, especially in immunocompromised patients.

LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

The ALADIN score can predict how well patients with alopecia areata will respond to JAK inhibitor treatments.

Profilin1 speeds up wound healing.

Immunological treatment improved both neuropathy and alopecia.

Litfulo is recommended for treating severe alopecia areata in people 12 and older, as it helps hair regrowth.

Mutations in the HEPHL1 gene cause abnormal hair and cognitive issues.

IL-17-producing γδ T cells help improve bone healing.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Targeting PTEN can improve healing in venous leg ulcers.

The CRH/CRH-R1 system might be involved in causing lichen planus.

The Fas/FasL pathway may play a role in alopecia areata.

Impaired LEF1 activation speeds up skin cell development in Hutchinson-Gilford Progeria Syndrome.

Patients with alopecia areata have higher levels of certain immune markers, suggesting new treatment targets.

iNKT cells help develop and maintain healthy skin in young mice.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.

Two mouse mutations cause similar hair loss despite different skin changes.