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Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Matriptase is crucial for skin, hair, and immune cell health, and its imbalance can lead to cancer.

Polymers can be designed to mimic natural cell environments for medical uses.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

3D images of skin show collagen is evenly spread, but elastic fibers are fewer near hair follicles.

Mimicking fetal wound environments may enable scarless healing in adults.

Targeting specific cell interactions may help treat skin fibrosis.

Benign follicular mucinosis involves immune cells attacking hair follicles.

Microporous scaffolds speed up skin healing and regeneration.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

Disulfide bonds make keratin in hair stronger and tougher.

Targeting macrophages may improve wound healing.

A unique skin cell similar to hair bulb melanocytes was identified, with better preservation using permanganate fixation.

Oxidized trichocyte keratin has a helical dislocation in its structure.

The hydrogel helps bone growth and healing in jaw and facial defects.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Endothelin-1 helps amelanotic melanocytes stick and move better on certain proteins.

The subcutaneous fascia is key to fast wound healing and could improve treatments for chronic wounds and scarring.

Changes in hair follicle structure may help diagnose and monitor alopecia.

Arg1+ macrophages may play a role in Alopecia Areata, offering new treatment targets.

The scaffold effectively prevents melanoma relapse and aids wound healing.

MOF controls skin development by regulating genes for mitochondria and cilia.

Gamma-irradiated amniotic fluid improves healing and reduces thickness of hypertrophic scars.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

A new tissue adhesive helps wounds heal better by allowing more cells to enter.

The ventral matrix is the main source of the nail plate.