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Anti-IL-12/IL-23 antibody therapy effectively treats psoriasiform skin lesions in IBD patients.

The study created special nanoparticles that effectively deliver an anti-inflammatory drug to treat skin inflammation in psoriasis.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

Removing UBE2N from skin cells causes inflammation and immune response, which can be lessened with specific inhibitors.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

TEC kinases may help cause inflammation in vitiligo and could be targeted for treatment.

KRT17 may be a new target for endometrial cancer treatment because it helps cancer cells move and form new blood vessels.

HDAC4 and HDAC7 are crucial for Th17 cell development and could be targeted to treat inflammatory diseases.

Keratin 17 is important for skin development and may help define skin cell types.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

Blocking IL-8 can reduce skin rashes from cancer treatment.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

ASLAN004 was safe and well-tolerated, supporting further development for treating certain diseases.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

Cholecystokinin may help reduce skin inflammation in psoriasis.

Stem cell vesicles reduced eczema symptoms in mice safely.

ILC1-like cells can cause alopecia areata by themselves.

A humanized IL-2 fusion protein boosts T regulatory cells and helps control hair loss in Alopecia Areata.

PX-12 may help treat psoriasis by blocking inflammation and cell death.

Dimethyl fumarate speeds up wound healing in IL-36Ra deficient mice by reducing NET formation and oxidative stress.

Blocking certain immune signals can reduce skin damage from radiation therapy.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

Inhibiting IL-17 and IL-23 improves wound healing in obese, diabetic mice by promoting healing macrophages.

AD-derived keratinocytes effectively mimic inflammation in atopic dermatitis.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Keratins 6, 16, and 17 increase in damaged or diseased skin and may help diagnose skin issues.

Targeting Keratin 17 may help overcome cancer therapy resistance.