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Activating cannabinoid receptor 1 may help manage psoriasis by reducing certain keratins.

Skin can heal wounds without hair follicle stem cells, but it takes a bit longer.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

The HCR gene contributes to psoriasis risk.

Targeting impaired Nrf2 signaling might help treat hidradenitis suppurativa early on.

Diseased horse foot skin shows increased keratin expression, similar to wound healing in mammals.

A new keratin gene was found in mice, explaining hair growth.

Epithelial proliferation and hyperkeratosis are linked to gastric ulcers in pigs.

Different parts of the nail express different keratins, showing unique patterns of differentiation.

Hair follicle stem cells can return to their original niche and help regenerate hair.

Human melanocytes in skin and hair follicles don't express keratin 16 or 6 naturally.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

Without keratin 10, there's more growth and development of oil-producing skin cells.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Suppressing ODC activity reduces tumor growth in hair follicles.

KAP6 genes are conserved across species and active in hair follicles.

Keratin K6irs is a marker for the inner root sheath of hair follicles in mice and humans.

A new method creates unique chemical structures that could improve drug discovery.

Keratin 6A increases skin inflammation, suggesting it could be a target for treating certain skin diseases.

S100A6 is important for cell functions and can help diagnose and treat diseases.

S100A6 protein is linked to disease progression, especially in cancers.

Two new proteins, hKAP1.6 and hKAP1.7, are found in the hair follicle cortex.

Keratin-associated proteins help link filaments and affect keratin's strength.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

The K5 promoter controls gene expression in skin cells, with specific DNA segments crucial for targeting and regulation.

The human K5 promoter controls specific gene expression in skin cells, with key regulatory elements near the TATA box.

Keratin-associated proteins are crucial for hair strength and structure.

IGF2BP3 gene is up-regulated in keloid patients, suggesting potential targets for treatment.