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Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.

CXCR2 in skin cells promotes tumor growth.

Rhodamin B stain is inconsistent for keratin in skin samples.

Mrp3 may aid in wound healing and hair growth.

ATR12-351 ointment safely delivers LEKTI protein to the skin, reducing enzyme activity in Netherton syndrome.

Plakophilin 1 helps control skin cell immune responses to prevent excessive inflammation.

Mrp3 helps in wound healing and hair growth.

Keratin 19 helps identify skin stem cells, with its presence varying by body location, age, and culture stage.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

The human K5 promoter controls specific gene expression in skin cells, with key regulatory elements near the TATA box.

The KRTAP gene family helps understand hair evolution and hair disorders.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

The HCR gene contributes to psoriasis risk.

Three specific genetic variants cause severe skin issues in children with EBS, highlighting the need for early genetic screening.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

LHX2, with other markers, can identify hair placodes in rats.

Keratin 6A increases skin inflammation, suggesting it could be a target for treating certain skin diseases.

The QuantAnts machines can find cancer markers and create CRISPR targets for them.

Hirosaki hairless rats lack hair due to missing DNA with key keratin genes.

Radiation damages hair stem cells and changes keratin expression, with Krt5 as a potential marker for radiation effects.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

New gene variants linked to a rare inherited hair loss disorder were found in three Chinese families.

The hairy ear mutation in mice is linked to changes in gene expression affecting hair growth.