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Certain genes are more active during wound healing in axolotl and Acomys, which could help develop materials that improve human wound healing and regeneration.

Neutrophils quickly move to the site of skin injury.

Scaffoldin helps form hard skin structures in chicken embryos.

PLIN2 affects hair growth in cashmere goats, potentially improving cashmere quality.

RNase L hinders hair growth by altering immune signals.

β‐thymosins, especially thymosin β4, help in heart protection, wound healing, and hair growth by stabilizing actin in cells.

Lymphotoxin-β is crucial for proper skin development in embryos.

lncRNAs are important for understanding and treating skin diseases.

CD61 is important for mouse tooth cell growth and works through Lgr5.

Tenascin-C and tenascin-W help control stem cell movement and growth in whisker follicles.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.

Activins and follistatins, part of the TGFβ family, are crucial for hair follicle development and skin health, affecting growth, repair, and the hair cycle.

LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

The scaffold improves wound healing and tissue regeneration.

Activin A helps heal skin wounds and protects the brain after injury.

TCF/Lef1 is essential for skin barrier function by regulating lipid metabolism.

Weak cell junctions disrupt hair follicle stem cell rest.

Aligned membranes improve wound healing by reducing scars and promoting skin regeneration.

Vimentin is crucial for wound healing, cell growth, and managing immune responses.

Paneth cells help support stem cells and aid tissue regeneration after injury.

Slug is crucial for skin health, hair growth, and healing.

The new patch made of cell matrix and a polymer improves wound healing and supports blood vessel growth.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

ECM components regulate β-Catenin activity, affecting wound healing.

Desmoglein 3 organization in cell connections changes without calcium, affecting cell adhesion.

DMBT1 and galectin-3 may help suppress epithelial skin cancer.

Three genes, BMP4, POSTN, and WNT5A, may help treat keloids.

Heparin-functionalized nanofabrics help heal wounds effectively and safely without scars in 14 days.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.