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The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Mature hair surfaces are formed by keratinized cells with developed layers, not just modified plasma membranes.

FN nanofiber dressings improve wound healing and restore natural skin structure.

Targeting immune pathways like JAK/STAT may help treat frontal fibrosing alopecia.

Hair curvature in Japanese people is linked to specific cell types and filament arrangements in the hair cortex.

Myofibroblasts in rat wound healing may come from blood vessel pericytes and perifollicular dermal sheath cells.

Certain proteins help nerve cells branch, and other findings relate to cancer, stem cell behavior, and cell division.

Macrophages help break down collagen around hair follicles during hair growth.

A new laser-based microscope can clearly image biological structures without labels.

Researchers found four distinct fibroblast types in human skin, which could help in treating wounds and fibrotic diseases.

Collagen makes skin stiff, and preservation methods greatly increase tissue stiffness.

Fibroblast spheres can form stem cells, but marker distribution needs more study.

Mimicking fetal wound environments may enable scarless healing in adults.

Hair tissue can help monitor environmental and public health risks.

Researchers found a new early sign of Frontal Fibrosing Alopecia that could help avoid misdiagnosis.

Trichohyalin is a versatile protein involved in hair and skin structure.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

Reactive lipids from aging cells change the extracellular matrix, affecting cell function and inflammation.

The connective tissue around hair follicles changes structure throughout the hair cycle.

AFM can diagnose hair disorders by revealing detailed hair surface changes.

Collagen fibrils in mink skin change structure during hair growth, becoming looser and thicker in the active phase.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Trichohyalin is found in non-hair tissues and works with filaggrin in certain skin areas and conditions.

Rat vibrissae structure relates to their sensory function.

Marsupial hair structure and keratin distribution are similar to placental mammals.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

Fibroblasts play a crucial role in the body's response to implants, needing more research for better treatments.

Hair structure forms the same way inside and outside the body.

A new tool helps study hair follicle cells to develop better treatments for hair disorders.