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Treating vitiligo with stem cells and melanocytes from hair, along with UVB light, works better than without the light.

Low oxygen levels improve the function of hair and skin cells when they are in direct contact.

Low fucosylation boosts stem cell growth in the eye.

KIT's role in skin cells is not entirely independent, as other cells can influence its function.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

Oxygen levels affect hair growth and color cells differently when they interact directly.

Hair graying happens because certain cells aren't maintained properly.

A unique skin cell similar to hair bulb melanocytes was identified, with better preservation using permanganate fixation.

The immune system can cause permanent skin and hair whitening by attacking pigment cells.

Researchers found three types of melanocytes in developing mouse skin, each with different genes and locations.

Transplantation is effective for stable leucoderma but not for progressive, widespread vitiligo vulgaris.

Human melanocytes have unique traits that affect melanoma development and prognosis.

Oncogenic melanocyte stem cells can develop into melanoma similar to human cases.

CD34+ and CD34- melanocyte stem cells have different regenerative abilities.

Sox9 is crucial for hair follicle stem cells to become melanocytes instead of glial cells.

Melanocyte stem cells hold promise for skin regeneration and treating pigmentation issues.

Melanocyte stem cells are crucial for skin pigmentation and have potential in disease modeling and regenerative medicine.

Immune cells boost stem cell activity in hairy moles, causing more hair growth.

Melanocyte stem cells in hair follicles are key to understanding and potentially preventing hair graying.

Exosomal miR-222-3p reduces melanin production in rabbits by targeting the SOX10 gene.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Stress can cause hair to turn gray by depleting stem cells.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

Understanding how melanocyte stem cells work could lead to new treatments for hair graying and skin pigmentation disorders.

The technique can isolate cells to help treat skin pigmentation issues.

Lack of a certain enzyme causes hair to grey early by damaging stem cells, but an antioxidant can help prevent this.

BMP4 helps stem cells turn into pigment-producing cells, affecting hair color and growth.

Melanocyte stem cells from non-affected skin in vitiligo patients can become functional melanocytes for potential therapy.

Hair follicle stem cells prevent melanocyte stem cells from differentiating by controlling retinoic acid levels.