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The document concludes that a systematic approach is crucial to identify causes of androgen excess in women beyond the most common cause, Polycystic ovary syndrome (PCOS).

Affected males are born with ambiguous genitalia, raised as females, but develop male traits at puberty due to enzyme deficiency.

Individuals with this condition often develop male traits and identities at puberty despite being raised as females.

High androgen levels can cause excessive hair growth and may indicate serious health issues, including heart disease and fertility problems.

Potential androgen excess signs vary by demographics and health, needing inclusive evaluation.

Masculinization in affected individuals occurs gradually after puberty due to hormone changes.

Too much male hormone is the main cause of Polycystic Ovary Syndrome.

Many young women who donate blood have hormonal disorders like excess male hormones and PCOS.

Exposure to fatty acids significantly increased androgen levels in female dogs, suggesting a link to conditions like insulin resistance and PCOS.

Male children's genital development issues can be caused by genetic mutations or environmental factors affecting hormone action.

Women with androgen excess, especially those with PCOS, have a much higher risk of heart disease and stroke.

High levels of testosterone and anti-Müllerian hormone before pregnancy are linked to a higher risk of newborns needing intensive care.

Adrenarche increases adrenal androgens around age 6, affecting hair growth and development.

The woman's symptoms and tests suggest her adrenal glands are producing too many male hormones.

A woman's hyperandrogenism was caused by a genetic mutation leading to non-classic adrenogenital syndrome.

Evaluate and manage androgen excess in women with a systematic approach, considering both physical and psychological impacts.

Most women with excess male hormones have Polycystic Ovary Syndrome, and hormonal therapy can improve symptoms but may cause side effects.

Oestrogen does not affect adrenal androgen levels in children and adolescents.

Reduced neurosteroids and growth issues can harm fetal brain development, especially in males.

New genes and pathways are important for testosterone production and male sexual development.

Genetic defects in androgen production are linked to male developmental disorders and are improving treatment understanding.

Androgen levels increase with puberty and are linked to sexual hair development.

Postmenopausal hyperandrogenism involves excess male hormones causing symptoms like hair growth and acne, and requires ruling out tumors and other disorders.

Women with congenital adrenal hyperplasia showed no prostatic growth.

Two women with very high androgen levels had only slight skin issues, one due to a non-classical adrenal disorder and the other due to an adrenal tumor.

25% of premenopausal women with Type 1 diabetes have undiagnosed androgen excess disorders.

Testosterone changes important cell communication proteins in pregnant rats' uteruses, which might affect pregnancy success.

Women with hyperandrogenism have higher androgen levels and lower SHBG, which may contribute to conditions like excessive hair growth and early puberty.