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Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Rural residents have higher vitamin D levels than urban residents in Erbil.

Deucravacitinib may help treat various immune diseases beyond psoriasis, but more research is needed.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

AKR1D1 controls glucocorticoid levels and receptor activity in liver cells.

The AVET system effectively delivers genes to human keratinocytes and may help treat skin diseases.

A man developed a rare skin condition after a hair transplant surgery.

Deucravacitinib led to full hair regrowth in a severe alopecia areata patient.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

RCVS should be considered in thunderclap headaches, and MRA and DSA are better for diagnosis than CT angiogram.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

DKK4 can be used to improve wool quality in Zhexi Angora rabbits.

The new ddPCR method reliably detects unwanted viruses in CAR-T cell products, ensuring their safety for patients.

Young patients with Vogt-Koyanagi-Harada syndrome may have poor vision outcomes despite treatment due to frequent recurrences.

Vogt-Koyanagi syndrome often leads to partial or complete blindness and responds poorly to treatment.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Vogt–Koyanagi–Harada disease can cause rare hair growth on normally hairless thumb skin.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Antiviral drugs, especially daclatasvir, may be a new treatment for a rare skin disease, improving survival and reducing symptoms in mice.

The research found that chickenpox virus spreads in skin through blood then to skin cells, while shingles virus moves from nerves to hair follicle areas before infecting skin cells.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

VDC-1101 shows potential as a treatment for canine cutaneous T-cell lymphoma.