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The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

The mTurq2-Col4a1 mouse model shows that cells can divide while attached to stable basement membranes during development.

KLHL24-mutant stem cells help understand skin and heart disease.

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Laminin-511 is essential for proper melanocyte movement and development in mice.

A new mouse mutation causes skin and hair issues, influenced by another gene.

The ACTH/MC2R system is crucial for controlling hair growth cycles in mice.

Combining hair transplantation with PRP is more effective for treating hair loss than hair transplantation alone.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Injecting α-MSH made mice's hair turn black.

Human placenta hydrolysate reduces inflammatory pain and nerve damage in mice.

CS12192 effectively treats alopecia areata with better safety than current options.

Stem cells slowed lung tumor growth but increased colon tumor growth in mice.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Replacing defective mesenchymal cells with normal ones fixes thymic growth issues in 22q11.2DS mouse models.

Collagen XVII and CD151 affect cell movement, with CD151 inhibiting migration when bound to integrins.

Hairless protein reduces Msx2 gene activity, affecting hair follicle development.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

A new mouse mutation causes skin and hair defects due to a gene change.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

A rare genetic disorder causes sparse hair and vision loss due to a CDH3 gene mutation.

A specific RNA increases hair stem cell growth and skin healing by affecting a protein through interaction with a microRNA.

Oncogenic melanocyte stem cells can develop into melanoma similar to human cases.

Chemical lumbar sympathectomy is an effective and safe treatment for idiopathic livedo reticularis.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.