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Urotensin II increases growth and VEGF production in rat skin cells by turning on the Wnt-β-catenin pathway.

LHX2, with other markers, can identify hair placodes in rats.

LHX2 is crucial for development, tissue repair, and preventing diseases.

The AVET system effectively delivers genes to human keratinocytes and may help treat skin diseases.

TGF-β3 controls stem cell growth and development, varying by cell type and conditions.

Keratins help GLUT2 stay on the cell membrane in pancreatic β-cells.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

VEGF and microvessel density are closely linked and peak during specific hair growth phases in cashmere goats.

Vimentin and keratin help viruses spread and could be targets for antiviral treatments.

Certain gene variations and different levels of BDNF and CRH hormones are linked to vitiligo.

Early recognition and treatment of VATS in transplant patients improve outcomes.

Keratin 17 is crucial for normal hair growth by regulating hair cycle transitions with TNFα.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Fibroblast growth factors could be a better, safer treatment for hair loss than current options.

Hair follicle cells produce VEGF, which can promote blood vessel cell growth and movement.

The VDR gene polymorphism does not affect BDNF levels in autoimmune thyroiditis and hypothyroidism patients.

Collagen-heparin-FGF2-VEGF scaffolds can improve skin healing.

FOXN1 duplication can cause excessive hair growth.

GPC1 is important for blood vessel growth in hair follicles and could help treat hair loss.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

The new ddPCR method reliably detects unwanted viruses in CAR-T cell products, ensuring their safety for patients.

The angora mouse mutation causes long hair and hair defects due to a gene deletion.

Tofacitinib is a promising treatment for children with rheumatic diseases.

Aδ-LTMRs have complex synapses with glycine, while Aβ-LTMRs have simpler ones.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

Plasma Rich in Growth Factors may help reduce hair loss in Frontal Fibrosing Alopecia.