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IL-17 and certain immune cells are linked to more severe alopecia areata.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

IL-18 signaling helps mature Tregs move into the thymus.

Blocking IL-17 can reduce joint inflammation in Ross River Virus infections.

Blocking IL-17 signaling may reduce skin inflammation and delay aging.

γδ T cells help control tissue scarring and blood vessel growth in response to foreign objects.

Tph cells are linked to the severity of systemic lupus erythematosus.

Keratin 17 is linked to various diseases, including cancer and skin conditions, and may be a target for diagnosis and treatment.

Anti-IL17A therapy reduced venous leg ulcer size by 64% without major side effects.

Blocking IL-17 signaling can delay skin aging and improve skin and hair health.

A specific group of immune and skin cells may cause chronic inflammation in atopic dermatitis.

IL-13 protein is much higher in the skin of atopic dermatitis patients than in healthy skin.

Psoriasis patients' immune response to a hair protein depends on their specific gene type.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

Targeting Vδ1+T-cells may help treat alopecia areata.

Type-2 immunity may influence skin diseases and could be targeted for treatment.

IL-17 inhibitors are more effective than methotrexate in preventing psoriatic arthritis in psoriasis patients.

Patients with alopecia areata have higher levels of certain immune markers, suggesting new treatment targets.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Higher Interleukin 17A levels may indicate more severe alopecia areata.

Adipocytes in atopic dermatitis skin change and worsen inflammation and fibrosis.

Higher IL-17A and IL-23 levels are linked to alopecia areata severity and could help in tracking and treating the disease.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

Psoriasis may be chronic because it lacks certain immune system controls that prevent overreaction.