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Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

IL-17-producing γδ T cells help improve bone healing.

γδ T cells help control tissue scarring and blood vessel growth in response to foreign objects.

Alopecia areata involves unique activation of certain immune cells.

Regulatory T cells protect hair follicle stem cells by maintaining immune privilege in the skin.

Targeting Vδ1+T-cells may help treat alopecia areata.

Keratin 17 is linked to various diseases, including cancer and skin conditions, and may be a target for diagnosis and treatment.

γδ T cells adapt uniquely to different tissues in mice.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

CD8+ T cells drive alopecia areata, while regulatory T cells are protective.

Different γδ T cell types have unique roles in causing alopecia areata.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Enhancing Tregs can protect against alopecia areata.

T cells with memory features grow in number and gather around hair follicles when there are not enough immune cells.

CD4 T cells need IFN-γ to cause hair loss in alopecia areata.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Patients with alopecia areata have higher levels of certain immune markers, suggesting new treatment targets.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Regulatory T cells and mesenchymal stem cells work together to prevent immune system overreactions and tissue damage.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.