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Researchers developed a new method to clearly see and label hair proteins with minimal errors using advanced freezing and microscopy techniques.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Adult hair follicle cells can create new hair follicles from corneal cells with the right support.

Trichoscopy is a quick, noninvasive method to diagnose hair and scalp disorders, often reducing the need for biopsies.

Trichoscopy is a useful and affordable tool for diagnosing Alopecia areata.

Human sweat glands contain stem cells capable of self-renewal and forming different cell types.

Notch pathway activation causes abnormal hair layer development.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Trichoscopy is a useful tool for diagnosing and managing hair and scalp conditions, often better than traditional methods.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Children with alopecia areata have different trichoscopic features than adults, including more empty follicular openings and pigtail hairs.

Blocking TGFβ-RI signaling enhances surface ectoderm differentiation from human stem cells.

Epidermal β-catenin activation changes the dermis by signaling different fibroblast types.

ZPK helps skin cells mature and may affect skin health.

K15 gene is mainly active in the basal layers of hair follicles and epithelia, aiding early skin cell development.

Trichoscopy helps diagnose and monitor the treatment of fungal scalp infections in children.

Recognizing trichoscopic features is crucial for diagnosing various hair loss conditions.

Trichofolliculoma was found in a person with amniotic band syndrome for the first time.

Hair shaft changes may be linked to CCCA, but their role is unclear.

Deep phenotyping helps distinguish between xeroderma pigmentosum and trichothiodystrophy, aiding in diagnosis and treatment.

The 4C32 gene may help in mouse skin development and differentiation.

Keratinocytes from dog hair follicles can create a functional skin layer in a lab model, useful for dog skin therapy.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

Trichothiodystrophy is a rare genetic disorder causing hair issues and sometimes linked to DNA repair defects.

Trichothiodystrophy is a condition with brittle hair and various physical and mental issues due to low sulfur in proteins.

Lgr5+ stem cells do not cause skin tumors.