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Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

Boosting certain cell signals can prevent hair loss from cancer treatments.

Skin cancer often starts from Lgr5+ progenitor cells.

Activating certain hair follicle cells could prevent hair loss from cancer treatments.

Krt15+ cells in the mouse intestine resist radiation and can start tumors.

Sporadic trichoblastic neoplasms generally don't recur or spread, with one case showing a specific genetic fusion.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Heat shock proteins help basal cell carcinoma grow by responding to inflammation signals.

HPV8 causes skin cancer by expanding specific skin stem cells.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

ErbB2 signaling is crucial for skin cell growth and cancer development in mice.

Long-standing benign tumors can become cancerous, especially in people with weakened immune systems.

Activating certain cells in hair follicles can prevent hair loss caused by cancer treatments.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Immortalized rat dermal papilla cells can still induce hair growth.

The surrounding tissue plays a crucial role in the growth and spread of skin cancer.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Intralesional chemotherapy with 5-fluorouracil and methotrexate may worsen keratoacanthoma-type skin cancer in transplant patients.

The research shows that skin cancer likely originates from hair follicles and that certain cell populations expand to promote skin cancer growth.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.