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Wnt4 protein makes the outer skin layer thicker in burn wounds by turning on a specific healing pathway and loosening the connections between skin cells.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

The LEF-1 gene in cashmere goats was successfully cloned and analyzed, showing potential for improving cashmere production.

Proper control of β-catenin activity is crucial for development and preventing diseases like cancer.

LIPH mutations cause woolly hair in some Chinese people.

The near-naked hairless mutation causes hair loss but is not due to a mutation in the hairless gene itself.

A new gene causes hairlessness and skin cysts in rats.

A gene mutation in Lama3 is linked to a common type of hair loss.

Stromal cells significantly influence Wnt signaling in the intestine, affecting cell growth and health.

Truncating mutations in the C2orf37 gene cause Woodhouse–Sakati syndrome.

A special gene region controls the re-emergence of a primitive wool type in Merino sheep, improving their wool yield and adaptability.

A rare genetic mutation causes severe immune issues, hair loss, and nail problems.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

A new mutation in the HR gene causes hair loss in a specific family.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Epidermal Wnt/β-catenin signaling controls fat cell formation and hair growth.

LEF-1 boosts cell growth in goat hair follicles, aiding cashmere production.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Scientists found gene mutations that affect hair loss, skin stem cells, and skin disorders, and identified drugs that may help treat blood vessel and skin conditions.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

DKK2 and SOSTDC1 together are necessary for the normal timing of the first regression phase in the hair growth cycle.

The hairless (hr) gene is essential for normal hair follicle function and its mutation leads to hair loss.

KY19382 helps regrow hair and create new hair follicles.

The Wnt signaling pathway is essential for skin repair and healing.

A genetic variant in the KRT25 gene causes tightly curled hair.

Dkk genes evolved faster in birds and reptiles, affecting hair development functions.

Blocking the Wnt/β‐catenin pathway can speed up wound healing, reduce scarring, and improve cartilage repair.

FOXN1 is crucial for thymus development and immune response in Xenopus laevis.

Healing skin wounds in mice can create new hair follicles, and adjusting Wnt signaling could potentially reduce scarring and treat hair loss.