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Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

ATP-sensitive potassium channels are important for hair growth.

Poncirin is a promising inhibitor of Janus Kinase 3, potentially better than tofacitinib.

Mice with a mutated Dsg3 gene showed severe symptoms but not the typical blistering of pemphigus vulgaris.

Caspase-3 helps control cell growth and organ size by activating YAP.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Lower SMAD2/3 activation predicts more severe skin cancer.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

A specific molecular switch, driven by MAPK/ERK signaling, helps spiny mice heal wounds by regenerating skin instead of forming scars.

Removing Mediator 1 causes teeth cells to turn into hair cells.

p63 controls Satb1 to help skin develop properly.

There are two ways to start hair growth: one needs Stat3 and the other does not, but both need PI3K activation.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

CircAGK affects cell death in hair cells by controlling the miR-3180-5p/BAX pathway, which can lead to hair loss.

SGK1 is important in cancer growth and treatment resistance, and targeting it could improve therapies.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

The K5 promoter controls gene expression in skin cells, with specific DNA segments crucial for targeting and regulation.

DcR3 helps heal wounds and regrow hair by changing macrophages to a repair-focused type.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

CXCR2 in skin cells promotes tumor growth.

Changing protein kinase levels in pituitary cells affects calcium flow and beta-endorphin release.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

The enzyme pyruvate kinase M2 helps hair regrowth and could be a potential treatment for hair loss.

UC.145 may be a new biomarker for predicting gastric cancer.

Activating TLR3 boosts autophagy gene expression in skin cells.