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Excess vitamin A changes skin and hair follicle development and affects polysaccharide distribution.

Male children's genital development issues can be caused by genetic mutations or environmental factors affecting hormone action.

Epidermal growth factor disrupts hair and gland formation in bandicoots.

E211 G>A gene linked to lower risk of severe prostate cancer and hair loss.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Some chemicals in cosmetics may harm pregnant individuals, fetuses, and newborns, and should be avoided unless necessary.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Suppressing ODC activity reduces tumor growth in hair follicles.

Hydroxyl radicals cause hair follicle cell death during chemotherapy by reducing Bcl-2 protein levels.

A mother's diet at conception can cause lasting genetic changes in her child.

Mutations in specific genes cause certain congenital defects in dogs, aiding in understanding similar human diseases.

Nitrogen mustard was not very effective for hair regrowth in alopecia areata and has potential skin cancer risk.

Hidradenitis suppurativa may be more severe in people with Mediterranean fever gene mutations.

Hair growth phase in mice weakens certain immune responses.

Topiramate impairs gene transmission in zebrafish, while Ginkgo biloba reduces harmful effects of high β-catenin levels.

Arumanis mango rind extract and its fractions can kill breast cancer cells, especially the ethanol extract and ethyl acetate fraction.

Anti-cancer treatments can cause reversible hair loss, skin sensitivity, pigmentation changes, nail damage, and skin reactions, with a need for more research on managing these side effects.

Targeted anticancer therapies can cause severe side effects similar to traditional chemotherapy, but with different types.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

PTCH gene mutations contribute to basal cell carcinoma development.

Riboflavin 5′-phosphate (FMN) shows potential for treating androgen-related conditions but may be limited in treating prostate cancer.

HGF/SF increases skin melanocytes but doesn't change melanin type or amount.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

Checkpoint inhibitor therapy can cause skin issues, from mild rashes to severe reactions.

Chemotherapy causes hair loss, nausea, fatigue, and blood issues, but counseling can help manage these effects.

Stat3 affects skin cancer development by altering keratinocyte stem cell behavior.

Activating PKCα in skin causes cell death and inflammation through different pathways.

Researchers developed reliable models to predict how well certain compounds bind to androgen receptors, emphasizing the importance of atomic electronegativity.