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A new compound slows cancer cell growth and causes cell death by blocking cell cycle progression and increasing cell-damaging molecules.

p63 is essential for controlling epithelial stem cells and tissue health.

Different types of PPARγ are found in varying amounts in human skin and its parts, which could affect how skin treatments work.

The document concludes that ERBB2 mutations are common in extramammary Paget disease and may respond to systemic treatments like cancer immunotherapy.

Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

Antibodies help identify glycoproteins in normal skin and tumor cells.

S100A4 promotes aggressive ovarian cancer and is a potential treatment target.

Bim and Puma proteins are found in developing mouse hair follicles and are involved in more than just cell death.

The study found that p63 needs signals from morphogens to help skin cells differentiate properly.

PH-762 shows promise in treating skin cancer by effectively targeting and silencing PD-1 in tumors with minimal side effects.

Identical p53 gene mutations in different cancers suggest the need for careful treatment.

Plet-1 protein helps hair follicle cells move and stick to tissues.

RAGE is a potential target for melanoma treatment, but its effectiveness is uncertain due to variable expression levels.

TNFα, IFNγ, and Substance P significantly affect prolactin levels in human skin, suggesting new treatments for skin and hair conditions.

CXCR2 in skin cells promotes tumor growth.

Removing Gsdma1/2/3 genes reduces skin cell overgrowth by blocking a specific cell pathway.

HPH-15, a new compound, effectively reduces skin fibrosis in experiments without causing harm.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

GPC1 is important for hair growth by helping blood vessels form around hair follicles.

Activating PPAR-γ signalling can protect hair follicle stem cells from damage caused by chemotherapy.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

Mutations in the p63 gene affect skin adhesion, barrier integrity, and hair growth.

The Skin POMC System affects hair growth and skin responses to stress.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

Hedgehog signaling is crucial for hair development, cadherins affect cell adhesion, neutrophils play a role in skin lesions, and BP230 autoantibodies impact skin stability.

The enzyme PA-PLA1α is important for proper hair follicle development.