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A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

PLA2R1 overexpression harms kidney cells by stopping their growth cycle.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Blocking miR-27a increases sheep hair follicle stem cell growth and decreases cell death, which could help improve wool quality and treat hair loss.

The -866G/A polymorphism in the UCP2 gene is not linked to polycystic ovary syndrome.

The hair defect is due to abnormal inner root sheath keratinization.

PSAT1 is key for hair growth and stem cell function in cashmere goats.

LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

PBX1 helps reduce aging and cell death in hair follicle stem cells by decreasing DNA damage, not by improving DNA repair.

Suppressing the HGPS mutation may improve symptoms and suggest reversibility.

Scientists can mimic hair disorders by altering genes in lab-grown human hair follicles, but these follicles lack some features of natural ones.

A gene mutation causes curly hair and hair loss in rats.

CaBP1 and 2 are necessary for maintaining calcium currents and hearing in inner ear cells.

OsUEV1B protein is essential for controlling phosphate levels in rice.

Impaired LEF1 activation speeds up skin cell development in Hutchinson-Gilford Progeria Syndrome.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Some people with schwannomatosis have a new type of mutation in the LZTR1 gene.

The CUBIC protocol allows detailed 3D visualization of proteins in mouse skin biopsies.

The Swedish neonatal screening program effectively detects PKU, galactosaemia, and biotinidase deficiency with low false positives.

Reduced TRPS1 leads to increased STAT3 and SOX9 in hair follicles, affecting hair growth.

The near-naked hairless mutation causes hair loss but is not due to a mutation in the hairless gene itself.

Deleting a specific protein in skin cells disrupts normal hair growth and development.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.

The patient's hair has unique structural differences with alternating bright and dark bands.

SPRY1 deficiency in skin cells causes stem cells to move to the skin surface, leading to increased pigmentation.

Removing alkaline phosphatase from human skin cells hinders the creation of new hair follicles.

A new mutation in the HR gene causes hair loss in a specific family.

The gene NM_026333 slows down aging by affecting the NCX1 pathway and could be targeted for anti-aging treatments.