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Keratin biomaterials from human hair help nerve regeneration by activating Schwann cells.

The Notch signaling pathway is important for hair follicle development and could help create treatments for hair disorders.

miR-22 helps skin cancer grow and spread by activating specific cell signals.

Fgf20 helps form hair follicle structures by stopping cell division and increasing cell movement.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Blocking EGFR in skin cells doesn't majorly increase inflammation markers.

Sphingosine 1-phosphate helps control mechanical pain.

Edar signaling is crucial for proper hair follicle development and function.

Follistatin helps hair growth and cycling, while activin prevents it.

Wnt/ß-catenin signaling is crucial for regulating skin stem cells and hair growth, with the right levels and timing needed for proper function.

The composite sponge helps heal diabetic wounds by reducing inflammation and promoting new blood vessel growth.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

KLHL24-mutant stem cells help understand skin and heart disease.

Keratin is crucial for skin barrier formation and affects mitochondrial function.

CCCA involves immune response and metabolism issues, suggesting new treatment options.

Genes linked to fibrosis are more active in people with central centrifugal cicatricial alopecia.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

Potential therapeutic targets for scarring hair loss are identified.

Endothelial TLR2 is crucial for timely wound healing, but HFSC TLR2 is not needed.

Allogenic ASCs and ECM transplants are safe and effective for tissue regeneration.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

A 3D cell model can rejuvenate stem cells to improve wound healing.

Rapamycin may help treat Leigh syndrome by targeting protein kinase C.

Fucoidan reduces bone cell formation by affecting T-cell activity.

A new ethical skin model using stem cells offers a reliable alternative for dermatological research.

Activin B improves wound healing and hair growth by helping stem cells move using certain cell signals.

Key genes can rewire networks, changing skin appendage types.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.