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The vitamin D receptor helps control hair growth and may protect against certain skin tumors.

Increasing Wnt5a in mice skin delays hair growth but doesn't stop it.

VDAC2 promotes cell death in cashmere goat hair follicles through the P53 pathway.

Ceramide Synthase 4 is essential for normal hair growth and preventing hair loss.

Weak cell junctions disrupt hair follicle stem cell rest.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

miR-22 helps skin cancer grow and spread by activating specific cell signals.

Cyclosporin promotes hair growth by boosting cell growth and reducing cell death.

Breast cancer patients taking CDK4/6 inhibitors are more likely to experience fatigue, hair loss, and mouth sores.

Wnt signaling is important for the change from the resting phase to the growth phase in human hair cycles.

Clitocybin A from a mushroom may promote hair growth by increasing cell activity in hair follicles.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

SOX18 helps sheep hair cells grow by activating a specific cell growth pathway.

Vitamin D and calcium are essential for effective wound healing and hair growth.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

Dock5 is important for skin healing and could help treat diabetic wounds.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

Wnt and SHH pathways help form hair follicles by coordinating cell processes.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

Activin B can boost hair growth by promoting cell proliferation and cell cycle progression.

Cyclosporine A may help treat hair loss by blocking a protein that inhibits hair growth.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Twist1 helps maintain important features of cells crucial for hair growth by working with Tcf4 and β-catenin.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

REV7 is crucial for genome stability and cancer treatment, making it a potential target for therapy.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

The hair follicle dermal sheath is essential for hair shedding and needs to communicate with the outer root sheath for normal hair growth cycles.

Injury changes how hair follicle stem cells behave, depending on the hair growth stage.